Eduardo E Benarroch

Summary

Affiliation: Mayo Clinic
Country: USA

Publications

  1. ncbi request reprint Involvement of medullary serotonergic groups in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Ann Neurol 55:418-22. 2004
  2. doi request reprint Signaling molecules of the CNS as targets of autoimmunity
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA Electronic address
    Handb Clin Neurol 133:17-38. 2016
  3. ncbi request reprint Depletion of ventromedullary NK-1 receptor-immunoreactive neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Foundation, Rochester, MN 55904, USA
    Brain 126:2183-90. 2003
  4. pmc Orthostatic intolerance without postural tachycardia: how much dysautonomia?
    Ajay K Parsaik
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Clin Auton Res 23:181-8. 2013
  5. pmc Loss of A5 noradrenergic neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 115:629-34. 2008
  6. ncbi request reprint Brainstem respiratory control: substrates of respiratory failure of multiple system atrophy
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, Minnesota 55905, USA
    Mov Disord 22:155-61. 2007
  7. ncbi request reprint Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis
    Shanu F Roemer
    Department of Neurology, Mayo Clinic, College of Medicine, 200 First St SW, Rochester, MN 55905, USA
    Brain 130:1194-205. 2007
  8. ncbi request reprint Involvement of medullary regions controlling sympathetic output in Lewy body disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 128:338-44. 2005
  9. ncbi request reprint Depletion of putative chemosensitive respiratory neurons in the ventral medullary surface in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Rochester, MN, USA
    Brain 130:469-75. 2007
  10. ncbi request reprint Involvement of hypocretin neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 113:75-80. 2007

Collaborators

Detail Information

Publications70

  1. ncbi request reprint Involvement of medullary serotonergic groups in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Ann Neurol 55:418-22. 2004
    ..Depletion of serotonergic neurons may contribute to impaired control of sympathetic outflow and other abnormalities in MSA...
  2. doi request reprint Signaling molecules of the CNS as targets of autoimmunity
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA Electronic address
    Handb Clin Neurol 133:17-38. 2016
    ....
  3. ncbi request reprint Depletion of ventromedullary NK-1 receptor-immunoreactive neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Foundation, Rochester, MN 55904, USA
    Brain 126:2183-90. 2003
    ....
  4. pmc Orthostatic intolerance without postural tachycardia: how much dysautonomia?
    Ajay K Parsaik
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Clin Auton Res 23:181-8. 2013
    ..In this study, we focussed on the question of how much dysautonomia, besides orthostatic tachycardia, is there in POTS vs. OI, and how the two groups compare in regards to clinical, autonomic, laboratory, and exercise variables...
  5. pmc Loss of A5 noradrenergic neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 115:629-34. 2008
    ..The severity of cell loss was comparable to that of locus ceruleus neurons in these cases. Our results indicate that A5 neurons are severely affected in MSA and this may contribute to some of the manifestations of this synucleinopathy...
  6. ncbi request reprint Brainstem respiratory control: substrates of respiratory failure of multiple system atrophy
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, Minnesota 55905, USA
    Mov Disord 22:155-61. 2007
    ..This finding may provide potential anatomical substrates for the respiratory manifestations of the disease...
  7. ncbi request reprint Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis
    Shanu F Roemer
    Department of Neurology, Mayo Clinic, College of Medicine, 200 First St SW, Rochester, MN 55905, USA
    Brain 130:1194-205. 2007
    ..These findings strongly support a role for a complement activating AQP4-specific autoantibody as the initiator of the NMO lesion, and further distinguish NMO from MS...
  8. ncbi request reprint Involvement of medullary regions controlling sympathetic output in Lewy body disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 128:338-44. 2005
    ..In LBD, orthostatic hypotension may be due primarily to involvement of sympathetic ganglion neurons rather than ventrolateral medulla neurons...
  9. ncbi request reprint Depletion of putative chemosensitive respiratory neurons in the ventral medullary surface in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Rochester, MN, USA
    Brain 130:469-75. 2007
    ..This may provide a possible morphological basis for impaired respiratory chemosensitivity and central sleep apnoea in this disorder...
  10. ncbi request reprint Involvement of hypocretin neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 113:75-80. 2007
    ..This is the first demonstration of loss of Hcrt/Orx neurons in MSA, which is consistent with a system degeneration of neurons involved in homeostatic function, including sleep and autonomic regulation, in this disorder...
  11. pmc Differential involvement of the periaqueductal gray in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, United States
    Auton Neurosci 158:111-7. 2010
    ..There was accumulation of glial cytoplasmic inclusions in all PAG columns in MSA. Our findings indicate involvement of the PAG columns in MSA, which may contribute to autonomic disturbances in this disorder...
  12. ncbi request reprint Differential involvement of hypothalamic vasopressin neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 129:2688-96. 2006
    ..Loss of AVP neurons in the SCN may contribute to impaired circadian regulation of endocrine and autonomic functions...
  13. doi request reprint Anhidrosis in multiple system atrophy involves pre- and postganglionic sudomotor dysfunction
    Elizabeth A Coon
    Department of Neurology, Mayo Clinic, Rochester, Minnesota, USA
    Mov Disord . 2016
    ....
  14. doi request reprint HCN channels: function and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:304-10. 2013
    ..Experimental evidence indicates that HCN channels may also contribute to mechanisms of epilepsy and pain. The physiologic functions of HCN channels and their implications for neurologic disorders have been recently reviewed.(1-10)...
  15. pmc Deconditioning in patients with orthostatic intolerance
    Ajay Parsaik
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 79:1435-9. 2012
    ..To study the frequency and degree of deconditioning, clinical features, and relationship between deconditioning and autonomic parameters in patients with orthostatic intolerance...
  16. doi request reprint Early seizure termination in ictal asystole
    Brian D Moseley
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Epilepsy Res 97:220-4. 2011
    ..4±13 s in IA, 67±28.9 s in ictal bradycardia, and 82.1±31.1 in non-bradycardic seizures. These were significantly different (ANOVA, p<0.02). This suggests cerebral hypoxia-ischemia favors seizure termination...
  17. pmc Autopsy confirmed multiple system atrophy cases: Mayo experience and role of autonomic function tests
    Valeria Iodice
    Neurovascular and Autonomic Medicine Unit, Imperial College London, UK
    J Neurol Neurosurg Psychiatry 83:453-9. 2012
    ..Published autopsy confirmed cases have provided reasonable neurological characterisation but have lacked adequate autonomic function testing...
  18. ncbi request reprint Glutamic acid decarboxylase autoimmunity with brainstem, extrapyramidal, and spinal cord dysfunction
    Sean J Pittock
    Department of Neurology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Mayo Clin Proc 81:1207-14. 2006
    ..To describe novel neurological manifestations associated with glutamic acid decarboxylase (GAD65) autoimmunity...
  19. pmc Ventilatory and cardiovascular responses to hypercapnia and hypoxia in multiple-system atrophy
    Axel Lipp
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Arch Neurol 67:211-6. 2010
    ..The cardiovascular responses to chemoreflex activation in MSA have not been explored to date...
  20. ncbi request reprint Preservation of branchimotor neurons of the nucleus ambiguus in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 60:115-7. 2003
    ..The number of neurons in the dorsal NA was not significantly different among the three groups. Neurons in the ventrolateral portion of the NA were depleted in MSA. Laryngeal stridor cannot be explained solely by neuronal loss in the NA...
  21. ncbi request reprint Brainstem respiratory chemosensitivity: new insights and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:2140-3. 2007
  22. doi request reprint Medullary neuronal loss is not associated with α-synuclein burden in multiple system atrophy
    Elizabeth A Coon
    Department of Neurology, Mayo Clinic, Rochester, Minnesota, USA
    Mov Disord 31:1802-1809. 2016
    ....
  23. doi request reprint Metabotropic glutamate receptors: synaptic modulators and therapeutic targets for neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:964-8. 2008
  24. ncbi request reprint Depletion of mesopontine cholinergic and sparing of raphe neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 59:944-6. 2002
    ..001) and laterodorsal tegmental nucleus (18 +/- 3 vs 47 +/- 4 cells/section, p < 0.001) in MSA. Whereas there was also depletion of locus ceruleus neurons, there was a striking preservation of rostral raphe neurons in MSA...
  25. ncbi request reprint EEG changes in a patient with steroid-responsive encephalopathy associated with antibodies to thyroperoxidase (SREAT, Hashimoto's encephalopathy)
    Alcibiades J Rodriguez
    Mayo Clinic Department of Neurology, Rochester, Minnesota, USA
    J Clin Neurophysiol 23:371-3. 2006
    ..These patients often show an excellent clinical improvement to immunosuppressive therapy, including corticosteroids. This case report documents the dramatic clinical and EEG improvement with steroid therapy...
  26. ncbi request reprint Dynamic phases of peroneal and tibial intraneural ganglia formation: a new dimension added to the unifying articular theory
    Robert J Spinner
    Department of Neurologic Surgery, Mayo Clinic, Rochester, Minnesota 55905, USA
    J Neurosurg 107:296-307. 2007
    ..Atypical features, which occasionally are observed, have offered an opportunity to verify further and expand on the authors' proposed theory...
  27. ncbi request reprint The ictal bradycardia syndrome: localization and lateralization
    Jeffrey W Britton
    Divisions of Epilepsy and Clinical Neurophysiology EEG, Department of Neurology, Mayo Clinic Rochester, Rochester, Minnesota 55905, USA
    Epilepsia 47:737-44. 2006
    ..However, the evidence for this is inconsistent. We sought to assess critically the predictable value of ictal bradycardia for seizure localization and lateralization...
  28. ncbi request reprint Paraventricular nucleus, stress response, and cardiovascular disease
    Eduardo E Benarroch
    Mayo Clinic, Dept of Neurology, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Clin Auton Res 15:254-63. 2005
    ..The neurochemical control of the different subgroups of PVN neurons is complex. The PVN has been implicated in the pathophysiology of congestive heart failure and the metabolic syndrome...
  29. doi request reprint Differential distribution of voltage-gated ion channels in cortical neurons: implications for epilepsy
    Nicholas D Child
    From the Department of Neurology, Mayo Clinic, Rochester, MN
    Neurology 82:989-99. 2014
    ....
  30. doi request reprint Orexin/hypocretin system and autonomic control: new insights and clinical correlations
    Daniela Grimaldi
    From the IRCCS Bologna Institute of Neurological Sciences D G, P C PRISM Laboratory A S and the Autonomic Unit P C, Department of Biomedical and Neuromotor Sciences, University of Bologna, Italy Department of Medicine D G, Sleep, Metabolism and Health Center, University of Chicago, IL and the Department of Neurology E E B, Mayo Clinic, Rochester, MN
    Neurology 82:271-8. 2014
    ..3-6) The present review focuses on the role of the orexin system in autonomic control and its potential implications in human NC. ..
  31. doi request reprint Microglia: Multiple roles in surveillance, circuit shaping, and response to injury
    Eduardo E Benarroch
    From the Department of Neurology, Mayo Clinic, Rochester, MN
    Neurology 81:1079-88. 2013
    ..Microglial dysfunction may also contribute to genetic neurobehavioral disorders, such as Rett syndrome. There are several comprehensive reviews on all these topics...
  32. pmc Clinical features and autonomic testing predict survival in multiple system atrophy
    Elizabeth A Coon
    1 Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Brain 138:3623-31. 2015
    ..Autonomic testing adds an additional, independent predictor of survival, demonstrating its value not only in the diagnosis of multiple system atrophy but also as prognostic marker...
  33. doi request reprint Histaminergic tuberomammillary neuron loss in multiple system atrophy and dementia with Lewy bodies
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, Minnesota, USA
    Mov Disord 30:1133-9. 2015
    ..All of these functions are affected in multiple system atrophy and dementia with Lewy bodies...
  34. ncbi request reprint Neocortical interneurons: functional diversity and clinical correlations
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 81:273-80. 2013
    ..There are many comprehensive reviews on GABAergic interneuron heterogeneity, development, plasticity, function in shaping cortical activity, and involvement in disease.(1-13.) ..
  35. doi request reprint Adenosine and its receptors: multiple modulatory functions and potential therapeutic targets for neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:231-6. 2008
  36. doi request reprint Suprachiasmatic nucleus and melatonin: reciprocal interactions and clinical correlations
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:594-8. 2008
  37. doi request reprint Descending monoaminergic pain modulation: bidirectional control and clinical relevance
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:217-21. 2008
  38. doi request reprint N-acetylaspartate and N-acetylaspartylglutamate: neurobiology and clinical significance
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:1353-7. 2008
  39. ncbi request reprint Aquaporin-4, homeostasis, and neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg or 811 Guggenheim Bldg, Rochester, MN 55905, USA
    Neurology 69:2266-8. 2007
  40. ncbi request reprint Tissue plasminogen activator: beyond thrombolysis
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:799-802. 2007
  41. ncbi request reprint Neurosteroids: endogenous modulators of neuronal excitability and plasticity
    Eduardo E Benarroch
    Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:945-7. 2007
  42. doi request reprint Brain glucose transporters: implications for neurologic disease
    Eduardo E Benarroch
    From the Department of Neurology, Mayo Clinic, Rochester, MN
    Neurology 82:1374-9. 2014
    ..1-7.) ..
  43. doi request reprint Pedunculopontine nucleus: functional organization and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:1148-55. 2013
    ..There have been several recent reviews on the PPN focused on its neurochemical organization and connectivity, physiology, involvement in parkinsonian syndromes, and as a target for DBS.(1-12.)..
  44. doi request reprint Insulin-like growth factors in the brain and their potential clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 79:2148-53. 2012
    ..The insulin-like peptides, their receptors, effects in the nervous system, and potential clinical correlations have been the subject of several recent reviews.(1-6)...
  45. ncbi request reprint Rho GTPases: role in dendrite and axonal growth, mental retardation, and axonal regeneration
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:1315-8. 2007
  46. ncbi request reprint Amphiphysin autoimmunity: paraneoplastic accompaniments
    Sean J Pittock
    Department of Neurology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Ann Neurol 58:96-107. 2005
    ..01). Overall, a minority of women (39%) and men (12%) had stiff-man phenomena. Only 10% of women (some with lung carcinoma) and 4% of men fulfilled diagnostic criteria for stiff-man syndrome...
  47. doi request reprint A PRESsing dissection
    Tamika M Burrus
    Mayo Clinic, Rochester, MN, USA
    Neurocrit Care 13:411-3. 2010
    ..When this occurs labile hypertension may be observed with its own complications, including but not limited to the clinico-radiographic entity, posterior reversible encephalopathy syndrome (PRES)...
  48. ncbi request reprint Brainstem in multiple system atrophy: clinicopathological correlations
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, Minnesota 55905, USA
    Cell Mol Neurobiol 23:519-26. 2003
    ..6. Loss of cholinergic mesopontine neurons, in the setting of loss of locus ceruleus neurons and preservation of rostral raphe neurons, may contribute to REM sleep abnormalities in MSA...
  49. doi request reprint Brain cholesterol metabolism and neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, MN 55905, USA
    Neurology 71:1368-73. 2008
  50. doi request reprint The midline and intralaminar thalamic nuclei: anatomic and functional specificity and implications in neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:944-9. 2008
  51. pmc Effect of position on valsalva maneuver: supine versus 20 degree position
    Elizabeth R Vogel
    Department of Neurology, Mayo Clinic, Rochester, Minnesota, USA
    J Clin Neurophysiol 25:313-6. 2008
    ..We recommend this modification for laboratory evaluation of the VM, whenever a flat-top response is seen...
  52. doi request reprint Subthalamic nucleus and its connections: Anatomic substrate for the network effects of deep brain stimulation
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 70:1991-5. 2008
  53. ncbi request reprint Thermoregulation: recent concepts and remaining questions
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:1293-7. 2007
  54. ncbi request reprint Lipid rafts, protein scaffolds, and neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg or 811 Guggenheim Bldg, Rochester, MN 55905, USA
    Neurology 69:1635-9. 2007
  55. ncbi request reprint Enteric nervous system: functional organization and neurologic implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 69:1953-7. 2007
  56. pmc Focal atrophy in dementia with Lewy bodies on MRI: a distinct pattern from Alzheimer's disease
    Jennifer L Whitwell
    Department of Radiology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 130:708-19. 2007
    ..Damage to this network of structures in DLB may affect a number of different neurotransmitter systems which in turn may contribute to a number of the core clinical features of DLB...
  57. ncbi request reprint GABAA receptor heterogeneity, function, and implications for epilepsy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:612-4. 2007
  58. ncbi request reprint Treatment of paroxysmal sympathetic hyperactivity
    Alejandro A Rabinstein
    Mayo Clinic, Department of Neurology, W8B, 200 First Street SW, Rochester, MN 55905, USA
    Curr Treat Options Neurol 10:151-7. 2008
    ....
  59. pmc Postural tachycardia syndrome: a heterogeneous and multifactorial disorder
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Mayo Clin Proc 87:1214-25. 2012
    ..Studies were limited to those published in English. Other articles were identified from bibliographies of the retrieved articles...
  60. ncbi request reprint New findings on the neuropathology of multiple system atrophy
    Eduardo E Benarroch
    Neurophysiological Laboratory, Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Auton Neurosci 96:59-62. 2002
    ....
  61. ncbi request reprint Clinical cues for detecting ictal asystole
    Gena R Ghearing
    Dept of Neurology, Mayo Clinic Rochester, Rochester, MN 55905, USA
    Clin Auton Res 17:221-6. 2007
    ..Recognition of ictal asystole is important as treatment with both antiepileptic drugs and cardiac pacing may be necessary. The purpose of this study was to identify clinical cues to aid in the detection of ictal asystole...
  62. doi request reprint TRP channels: functions and involvement in neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 70:648-52. 2008
  63. ncbi request reprint Neuron-astrocyte interactions: partnership for normal function and disease in the central nervous system
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic College of Medicine, 200 First St SW, Rochester, MN 55905, USA
    Mayo Clin Proc 80:1326-38. 2005
    ..Disturbances of these neuron-astrocyte interactions are likely to play an important role in neurologic disorders including cerebral ischemia, neurodegeneration, migraine, cerebral edema, and hepatic encephalopathy...
  64. ncbi request reprint Sodium channels and pain
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 68:233-6. 2007
  65. doi request reprint Neural control of the gastrointestinal tract: implications for Parkinson disease
    Maria G Cersosimo
    Parkinson s Disease and Movement Disorder Unit, Hospital de Clinicas, University of Buenos Aires, Argentina
    Mov Disord 23:1065-75. 2008
    ..This early involvement provides insights into the pathophysiology of gastrointestinal dysmotility in this disorder and may constitute an important step in the etiopathogenesis of Lewy body disease...
  66. ncbi request reprint Quantitative study of salivary secretion in Parkinson's disease
    Omar R Tumilasci
    Departamento de Fisiologia, Laboratorio de Neurofisiologia, Facultad de Medicina, and Programa de Parkinson y Movimientos Anormales, Instituto de Neurociencias del Hospital de Clínicas, Universidad de Buenos Aires, Buenos Aires, Argentina
    Mov Disord 21:660-7. 2006
    ..Domperidone may have a peripheral effect that potentiates reflex salivary secretion. Salivary composition is abnormal in PD and is not affected by levodopa treatment...
  67. ncbi request reprint Neurodegeneration involving putative respiratory neurons in Perry syndrome
    Yoshio Tsuboi
    Department of Neurology, Fukuoka University, Fukuoka, Japan
    Acta Neuropathol 115:263-8. 2008
    ....
  68. ncbi request reprint Neurovascular unit dysfunction: a vascular component of Alzheimer disease?
    Eduardo E Benarroch
    Neurology 68:1730-2. 2007
  69. ncbi request reprint Dopamine beta-hydroxylase deficiency involves the central autonomic network
    William P Cheshire
    Acta Neuropathol 112:227-9. 2006
  70. ncbi request reprint Autonomic and cognitive dysfunction in Parkinson's disease
    Juan Idiaquez
    Universidad de Valparaiso, Valparaiso, Chile
    Clin Auton Res 17:93-8. 2007
    ....