T Nakajima

Summary

Affiliation: Science University of Tokyo
Country: Japan

Publications

  1. ncbi request reprint Induction of ubiquitin conjugating enzyme activity for degradation of topoisomerase II alpha during adenovirus E1A-induced apoptosis
    T Nakajima
    Department of Biological Science and Technology, Science University of Tokyo, Noda, Japan
    Biochem Biophys Res Commun 239:823-9. 1997
  2. ncbi request reprint Stabilization of p53 by adenovirus E1A occurs through its amino-terminal region by modification of the ubiquitin-proteasome pathway
    T Nakajima
    Department of Biological Science and Technology, Science University of Tokyo, Noda 278, Japan
    J Biol Chem 273:20036-45. 1998
  3. ncbi request reprint A novel adenovirus E1B19K-binding protein B5 inhibits apoptosis induced by Nip3 by forming a heterodimer through the C-terminal hydrophobic region
    N Ohi
    Department of Biological Science and Technology, Science University of Tokyo, Noda, Japan
    Cell Death Differ 6:314-25. 1999
  4. ncbi request reprint Expression of various viral and cellular enhancer-promoters during differentiation of human embryonal carcinoma cells
    T Hasegawa
    Department of Applied Biological Science, Science University of Tokyo, Chiba, Japan
    Differentiation 42:191-8. 1990
  5. ncbi request reprint A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer
    M Suzuki
    Department of Biological Science and Technology, Science University of Tokyo, Chiba, Japan
    Oncogene 17:853-65. 1998

Collaborators

Detail Information

Publications5

  1. ncbi request reprint Induction of ubiquitin conjugating enzyme activity for degradation of topoisomerase II alpha during adenovirus E1A-induced apoptosis
    T Nakajima
    Department of Biological Science and Technology, Science University of Tokyo, Noda, Japan
    Biochem Biophys Res Commun 239:823-9. 1997
    ..These results indicate that a ubiquitination factor(s) specific to topo II alpha is induced during E1A-induced apoptosis in MA1 cells...
  2. ncbi request reprint Stabilization of p53 by adenovirus E1A occurs through its amino-terminal region by modification of the ubiquitin-proteasome pathway
    T Nakajima
    Department of Biological Science and Technology, Science University of Tokyo, Noda 278, Japan
    J Biol Chem 273:20036-45. 1998
    ..These results indicate that the stabilization of p53 by E1A occurs through modification of a ubiquitin-specific enzyme(s) in the ubiquitin-proteasome pathway...
  3. ncbi request reprint A novel adenovirus E1B19K-binding protein B5 inhibits apoptosis induced by Nip3 by forming a heterodimer through the C-terminal hydrophobic region
    N Ohi
    Department of Biological Science and Technology, Science University of Tokyo, Noda, Japan
    Cell Death Differ 6:314-25. 1999
    ..However the deletion mutant B5Delta1-31 lacking the N-terminus does induce apoptosis, although weaker than does Nip3, suggesting that the N-terminal region is masking the apoptosis-inducing capacity of B5...
  4. ncbi request reprint Expression of various viral and cellular enhancer-promoters during differentiation of human embryonal carcinoma cells
    T Hasegawa
    Department of Applied Biological Science, Science University of Tokyo, Chiba, Japan
    Differentiation 42:191-8. 1990
    ..These results indicate that factor(s) regulating activities of various enhancer-promoters in NEC14 cells is or are different from E1A-like factor(s) present in mouse EC F9 cells...
  5. ncbi request reprint A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer
    M Suzuki
    Department of Biological Science and Technology, Science University of Tokyo, Chiba, Japan
    Oncogene 17:853-65. 1998
    ..Although E2FBP1 lacks the transactivation domain, it stimulates E2F site-dependent transcription in cooperation with E2F-1...